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Med J Tabriz Uni Med Sciences Health Services. 2013;35(2): 90-97.
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Original Article

Lovastatin Enhances Paraoxonase Enzyme Activity and Quells low-density Lipoprotein Susceptibility to Oxidation in Type 2 Diabetic Nephropathy

Mohammad Naghavi Behzad 1, Nariman Nezami 2, Reza Piri 1, Javid Safa 2, Behzad Salari 2, Nadereh Rashtchi Zadeh 3, Amir Ghorbanihaghjo 2*

1 Research Center for Philosophy and History of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran
2 Drug Applied Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
3 Biotechnology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
*Corresponding Author: Email: ghorbaniamir@hotmail.com

Abstract

Background & Objectives: This study was done to investigate the effect of lovastatin therapy on paraoxonase 1 (PON1) and arylesterase (ARE) activities, and low-density lipoprotein cholesterol (LDL-C) susceptibility to oxidation in people with type 2 diabetic nephropathy (T2DN). & Methods: Lovastatin (20 mg/day) was administered to 30 people with T2DN for 90 days and then withdrawn for 30 days. PON1 and ARE activities were measured by the spectrophotometric method. Susceptibility of LDL-C to oxidation was determined as the production of conjugated dienes. Results: After 90 days of lovastatin intervention, PON1 and ARE activities and LDL-C lag phase were significantly increased (p=0.004, 0.002, and b0.001). After 30 days of lovastatin withdrawal, PON1 and ARE activities and LDL-C lag phase had not changed significantly. Conclusion: Lovastatin therapy improves PON1 and ARE activities, and LDL-C susceptibility to oxidation. Despite withdrawal of lovastatin, PON1 and ARE activities, and LDL-C susceptibility to oxidation remain unchanged for a long period of time in people with T2DN.
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Submitted: 09 Sep 2012
Accepted: 18 Dec 2012
ePublished: 26 Jun 2013
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